Viruses and Atherosclerosis [electronic resource] / by Anton Kutikhin, Elena Brusina, Arseniy E. Yuzhalin.

By: Kutikhin, Anton [author.]Contributor(s): Brusina, Elena [author.] | Yuzhalin, Arseniy E [author.] | SpringerLink (Online service)Material type: TextTextSeries: SpringerBriefs in Immunology ; 4Publisher: New York, NY : Springer New York : Imprint: Springer, 2013Description: X, 69 p. 1 illus. online resourceContent type: text Media type: computer Carrier type: online resourceISBN: 9781461488637Subject(s): Medicine | Immunology | Medical microbiology | Virology | Biomedicine | Virology | Immunology | Medical MicrobiologyAdditional physical formats: Printed edition:: No titleDDC classification: 616.9101 LOC classification: QR355-502Online resources: Click here to access online
Contents:
The Hypothesis of Virus-Driven Atherosclerosis -- The Role of Herpes Simplex Virus-1 and Herpes Simplex Virus-2 in Atherosclerosis -- How may HSV promote atherosclerosis? -- Evidence from epidemiological studies -- Evidence from cardiovascular events -- The Role of Epstein-Barr virus in Atherosclerosis -- EBV and atherosclerosis: the published data from basic and epidemiological investigations -- The Role of Enteroviruses, Parvovirus B19, Respiratory Syncytial Virus and Measles Virus in Atherosclerosis -- Enteroviruses -- Parvovirus B19 -- Respiratory Syncytial Virus (RSV) -- Measles Virus -- The Role of Hepatitis A, B, and C in Atherosclerosis -- Hepatitis A virus (HAV), atherosclerosis, and related diseases -- Hepatitis B virus (HBV), atherosclerosis, and related diseases -- Hepatitis C virus (HCV), atherosclerosis, and related diseases -- HCV and changes in immunological and atherotic profile -- HCV and subclinical atherosclerosis -- HCV and CAD -- HCV and myocardial infarction -- HCV and stroke -- Conclusions -- Summaries: Are We There Yet?.
In: Springer eBooksSummary: Will address an important, yet underrepresented, topic. The correlation between viruses and atherosclerosis has been a focal point of the authors’ work, for a number of years. This volume will explore the relationship between different viral strains and   atherosclerosis. It will begin by describing the hypothesis and denoting the mechanisms of virus-driven atherosclerosis, then expanding on the subject by focusing on different virus strains—from Herpes, to Epstein-Barr, to the triad of Hepatitis viruses, et al—on a chapter-by-chapter basis. While there are books, albeit few, that cover particular viral strains and their relationship to cardiovascular diseases, this work will be unique in its scope by considering multiple strains of viruses, making it a repository of information on the topic; a truly comprehensive volume.
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The Hypothesis of Virus-Driven Atherosclerosis -- The Role of Herpes Simplex Virus-1 and Herpes Simplex Virus-2 in Atherosclerosis -- How may HSV promote atherosclerosis? -- Evidence from epidemiological studies -- Evidence from cardiovascular events -- The Role of Epstein-Barr virus in Atherosclerosis -- EBV and atherosclerosis: the published data from basic and epidemiological investigations -- The Role of Enteroviruses, Parvovirus B19, Respiratory Syncytial Virus and Measles Virus in Atherosclerosis -- Enteroviruses -- Parvovirus B19 -- Respiratory Syncytial Virus (RSV) -- Measles Virus -- The Role of Hepatitis A, B, and C in Atherosclerosis -- Hepatitis A virus (HAV), atherosclerosis, and related diseases -- Hepatitis B virus (HBV), atherosclerosis, and related diseases -- Hepatitis C virus (HCV), atherosclerosis, and related diseases -- HCV and changes in immunological and atherotic profile -- HCV and subclinical atherosclerosis -- HCV and CAD -- HCV and myocardial infarction -- HCV and stroke -- Conclusions -- Summaries: Are We There Yet?.

Will address an important, yet underrepresented, topic. The correlation between viruses and atherosclerosis has been a focal point of the authors’ work, for a number of years. This volume will explore the relationship between different viral strains and   atherosclerosis. It will begin by describing the hypothesis and denoting the mechanisms of virus-driven atherosclerosis, then expanding on the subject by focusing on different virus strains—from Herpes, to Epstein-Barr, to the triad of Hepatitis viruses, et al—on a chapter-by-chapter basis. While there are books, albeit few, that cover particular viral strains and their relationship to cardiovascular diseases, this work will be unique in its scope by considering multiple strains of viruses, making it a repository of information on the topic; a truly comprehensive volume.

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